[1]Li XW,Liang SS,Le WB,et al.Long-term outcome of IgA nephropathy withminimal change disease:a comparison between patients with and without minimal change disease.J Nephrol,2016,29(4):567-573.
[5]Szeto CC,Lai FM,Chow KM,et al.Long-term outcome of biopsyproven minimal change nephritic in Chinese adult.Am J Kidney Dis,2015,65(5):710-718.
[6]Munyentwali H,Bouachi K,Audard V,et al.Rituximab is an efficient and safe treatment in adults with steroid-dependent minimal change disease.Kidney Int,2013,83(3):511-516.
[7]Brugnano R,Del Sordo R,Covarelli C,et al.IgM nephropathy:is it closer tominimal change disease or to focal segmental glomerulosclerosis?J Nephrol,2016,29(4):479-486.
[8]Kitiyakara C,Eggers P,Kopp JB.Twenty-one-year trend in ESRD due to focal segmental glomerulosclerosis in the United States.Am J Kidney Dis,2004,44(5):815-825.
[9]Bellur SS,Lepeytre F,Vorobyeva O,et al.Evidence from the Oxford Classification cohort supports the clinical value of subclassification of focal segmental glomerulosclerosis in IgA nephropathy.Kidney Int,2017,91(1):235-243.
[10]Daskalakis N,Winn MP.Focal and segmental glomerulosclerosis.Cell Mol Life Sci,2006,63(21):2506-2511.
[11]DAgati V,Fogo AB,Bruijin JA,et al.Pathologic classification of focal segmental glomerulosclerosis:A working proposal.Am J of Kidney Dis,2004,43(2):368-382.
[12]Pullen N,Fornoni A.Drug discovery in focal and segmental glomerulosclerosis.Kidney Int,2016,89(6):1211-1220.
[13]Buscher AK,Konrad M,Nagel M,et al.Mutations in podocyte genes are a rare cause of primary FSGS associated with ESRD in adult patients.Clin Nephrol,2012,78(1):47-53.
[14]Vivarelli M,Massella L,Ruggiero B,et al.Minimal Change Disease.Clin JAm Soc Nephrol,2017,12(2):332-345.
[16]Lowik MM,Groenen PJ,Pronk I,et al.Focal segmental glomerulosclerosis in a patient homozygous for a CD2AP mutation.Kidney Int,2007,72(10):1198-1203.
[17]NovelliR,Gagliardini E,Ruggiero B,et al.Any value of podocyte B7-1 as a biomarker in human MCD and FSGS?Am J Physiol Renal Physiol,2016,310(5):F335-F341.
[19]Gupta A,Quigg RJ.Glomerular Diseases Associated With Hepatitis B and C.Adv Chronic Kidney Dis,2015,22(5):343-351.
[20]Radice A,Trezzi B,Maggiore U,et al.Clinical usefulness of autoantibodies to M-type phospholipase A2 receptor(PLA2R)for monitoring disease activity in idiopathic membranous nephropathy(IMN).Autoimmun Rev,2016,15(2):146-154.
[21]Roccatello D,Sciascia S,Di Simone D,et al.New insights into immune mechanisms underlying response to Rituximab in patients withmembranous nephropathy:A prospective study and a review of the literature.Autoimmun Rev,2016,15(6):529-538.
[22]Lee T,Derebail VK,Kshirsagar AV,et al.Patients with primary membranous nephropathy are at high risk of cardiovascular events.Kidney Int,2016,89(5):1111-1118.
[23]Fogo AB,Lusco MA,Najafian B,et al.AJKD Atlas of Renal Pathology:Minimal Mesangial and Mesangial Proliferative Lupus Nephritis(ISN/RPSClassⅠandⅡ).Am J Kidney Dis,2017,70(2):7-8.
[24]Shin HS,Cho DH,Kang SK,et al.Patterns of renal disease in South Korea:a 20-year review of a single-center renal biopsy database.Ren Fail,2017,39(1):540-546.
[25]Presta P,Minutolo R,Iodice C,et al.Renin-angiotensin system inhibitors reduce the progression of mesangioproliferative glomerulonephritis:10 year follow-up.Eur J Intern Med,2011,22(6):90-94.
[26]Choudhry S1,Bagga A,Hari P,et al.Efficacy and safety of tacrolimus versus cyclosporine in children with steroid-resistant nephrotic syndrome:a randomized controlled trial.Am J Kidney Dis,2009,53(5):760-769.
[27]Fukuma Y,Hisano S,Segawa Y,et al.Clinicopathologic correlaction of C1q nephropathy in children.Am JKidney Dis,2006,47(3):412-418.
[28]Luo C,Chen D,Tang Z,et al.Clinicopathological features and prognosis of Chinese patients with acute post-streptococcal glomerulonephritis.Nephrology(Carlton),2010,15(6):625-631.
[29]Gunasekaran K,Krishnamurthy S,Mahadevan S,et al.Clinical Characteristics and Outcome of Post-Infectious Glomerulonephritis in Children in Southern India:A Prospective Study.Indian J Pediatr,2015,82(10):896-903.
[30]Eison TM,Ault BH,Jones DP,et al.Post-streptococcal acute glomerulonephritis in children:clinical features and pathogenesis.Pediatr Nephrol,2011,26(2):165-180.
[31]Aliel-TM,Babikir AM,El-Assad S,et al.Prognosis of acute poststreptococcal glomerulonephritis in Sudanese children.Arab J Nephrol Transplant,2014,7(2):103-107.
[32]Conti G,De Vivo D,Vitale A,et al.Dense deposit disease in a child with febrile sore throat.Saudi J Kidney Dis Transpl,2017,28(4):925-928.
[33]Rudnicki M.Rituximab for Treatment of Membranoproliferative Glomerulonephritis and C3 Glomerulopathies.Biomed Res Int,2017,2017(9):1-7.
[34]Suryawanshi M,Karnik S,Roy S.Clinicopathological Analysis of Glomerular Disease of Adult Onset Nephrotic Syndrome in an Indian Cohort-A Retrospective Study.JClin Diagn Res,2017,11(5):25-30.
[35]Ravindran A,Fervenza FC,Smith RJH,et al.C3 glomerulonephritis with a severe crescentic phenotype.Pediatr Nephrol,2017,32(9):1625-1633.
[36]Yurova VA,Bobrova LA,Kozlovskaya NL,et al.Changes in the complement system in membranoproliferative glomerulonephritis.Ter Arkh,2017,89(6):69-77.
[37]Han F,Chen L,Le J,et al.The Clinicopathologic Spectrum of Rapidly Progressive Glomerulonephritis Based on Glomerular Immune Deposition and Antineutrophil Cytoplasmic Antibody.Appl Immunohistochem Mol Morphol,2015,23(10):704-710.
[41]Choudhry S,Bagga A,Hari P,et al.Efficacy and safety of tacrolimus versus cyclosporine in children with steroid-resistant nephrotic syndrome:a randomized controlled trial.Am J Kidney Dis,2009,53(5):760-769.
[44]Hahn BH,McMahon MA,Wilkinson A,etal.American College of Rheumatology guidelines for screening,treatment,and management of lupus nephritis.Arthritis Care Res(Hoboken),2012,64(6):797-808.
[58]LS,Liu ZH.Epidemiologic data of renal disease from a single unit in China:analysis based on 13519 renal biopsies.Kidney Int,2004,66:920-923.
[59]Lau KK,Wyatt RJ,Moldoveanu Z,et al.Serum levels of galactose-deficient IgA in children with IgA nephropathy and Henoch-Sch?nlein purpura.Pediatr Nephrol,2007,22(12):2067-2072.
[60]Kamei K,Ogura M,Sato M,et al.Evolution of IgA nephropathy into anaphylactoid purpura in six cases—further evidence that IgA nephropathy and Henoch-Schonlein purpura nephritis share common pathogenesis.Pediatr Nephrol,2016,31(5):779-785.
[61]Jennette JC,Falk RJ,Bacon PA,et al.2012 Revised International Chapel Hill Consensus Conference nomenclature of vasculitides.Arthritis Rheum,2013,65(1):1-11.
[62]Sun LJ,Shan JP,CuiRL,et al.Combination therapy with lamivudine and angiotensin-converting enzyme inhibitor/angiotensin receptor blocker for hepatitis B virus-associated glomerulonephritis with mild tomoderate proteinuria:a clinical review of 38 cases.Int Urol Nephrol,2017,49(6):1049-1056.
[63]Salter T,Burton H,Douthwaite S,et al.Immune Complex Mediated Glomerulonephritis with Acute Thrombotic Microangiopathy following Newly Detected Hepatitis B Virus Infection in a Kidney Transplant Recipient.Case Rep Transplant,2016,2016:3152495.
[64]Sun YH,Lei XY,Sai YP,et al.Relationship between genotypes and clinical manifestation,pathology,and cccDNA in Chinese children with hepatitis B virus-associated glomerulonephritis.World JPediatr,2016,12(3):347-352.
[65]Wang C,Ye ZY,Zeng DH,et al.Clinicopathological features of cryoglobulinemic glomerulonephritis associated with HBV infection:a retrospective analysis of 8 cases in China.Int JClin Exp Pathol,2015,8(9):10475-10481.
[66]McAdoo SP,Pusey CD.Anti-Glomerular Basement Membrane Disease.Clin JAm Soc Nephrol,2017,12:1162-1172.
[67]Prabhakar D,Rathi M,Nada R,et al.Anti-glomerular basement membrane disease:Case series from a tertiary center in North India.Indian J Nephrol,2017,27(2):108-112.
[68]Liu JH,Wei XX,Li A,et al.Novel mutations in COL4A3,COL4A4,and COL4A5 in Chinese patients with Alport Syndrome.PLoS One,2017,12(5):e0177685.
[69]Nozu K,Minamikawa S,Yamada S,et al.Characterization of contiguous gene deletions in COL4A6 and COL4A5 in Alport syndrome-diffuse leiomyomatosis.JHum Genet,2017,62(7):733-735.
[70]Deng S,Xu H,Yuan J,et al.Identification of a novel collagen type IV alpha-4(COL4A4)mutation in a Chinese family with autosomal dominant Alport syndrome using exome sequencing.Indian JMed Res,2016,144(2):200-205.
[71]Murata T,Katayama K,Oohashi T,et al.COL4A6 is dispensable for autosomal recessive Alport syndrome.Sci Rep,2016,6:29450.
[72]Xu Y,Guo M,Dong H,et al.A Novel COL4A4 Mutation Identified in a Chinese Family with Thin Basement Membrane Nephropathy.Sci Rep,2016,6:20244.
[73]Deltas C,Savva I,Voskarides K,et al.Carriers of Autosomal Recessive Alport Syndrome with Thin Basement Membrane Nephropathy Presenting as Focal Segmental Glomerulosclerosis in Later Life.Nephron,2015,130(4):271-280.
[74]Chinen Y,Nakamura S,Yoshida T,etal.A new mutation found in newborn screening for Fabry disease evaluated by plasma globotriaosylsphingosine levels.Hum Genome Var,2017,4:17002.
[76]DiMartino MT,Scionti F,Sestito S,et al.Genetic variants associated with gastrointestinal symptoms in Fabry disease.Oncotarget,2016,7(52):85895-85904.
[77]Cohen AH.Collagen Type III Glomerulopathies.Adv Chronic Kidney Dis,2012,19(2):101-106.
[78]R?rtveit R,Reiten MR,Lingaas F,et al.Glomerular Collagen V Codeposition and Hepatic Perisinusoidal Collagen III Accumulation in Canine Collagen Type III Glomerulopathy.Vet Pathol,2015,52(6):1134-1141.
[79]Chen X,Wang H,Xu W,et al.Collagen type III glomerulopathy:case report and review of the literature.Clin Nephrol,2017,87(1):39-46.
[81]Ohtsubo H,Okada T,Nozu K,et al.Identification of mutations in FN1 leading to glomerulopathy with fibronectin deposits.Pediatr Nephrol,2016,31(9):1459-1467.
[82]Yoshino M,Miura N,Ohnishi T,et al.Clinicopathological analysis of glomerulopathy with fibronectin deposits(GFND):a case of sporadic,elderly-onset GFND with codeposition of IgA,C1q,and fibrinogen.Intern Med,2013,52(15):1715-1720.
[88]Mogensen CE,Schmitz O.The diabetic kidney:from hyperfiltration and microalbuminuria to end-stage renal failure.Med Clin North Am,1988,72(6):1465-1492.
[90]Wada J,Makino H.Historical chronology of basic and clinical researchin diabetic nephropathy and contributions of Japanese scientists.ClinExp Nephrol,2009,13(5):405-414.
[91]Nakagawa T,Tanabe K,Croker BP,et al.Endothelial dysfunction as apotential contributor in diabetic nephropathy.Nat Rev Nephrol,2011,7(1):36-44.
[95]Wahba IM,Mak RH.Obesity and obesity-initiated metabolic syndrome:mechanistic links to chronic kidney disease.Clin J Am Soc Nephrol,2007,2(3):550-562.
[96]Wu Y,Liu Z,Xiang Z,et al.Obesity-related glomerulopathy:insight from gene expression profiles of the glomeruli derived from renal biopsy samples.Endocrinology,2006,147(1):44-50.
[97]Maric-Bilkan C.Obesity and diabetic kidney disease.Med Clin North Am,2013,97(1):59-74.
[105]Fakhoufi F,Fremeaux-Bacchi V,Noel LH,et al.C3 glomerulopathy:a new classification.Nat Rev Nephrol,2010,6(8):494-499.
[106]Medjeral-Thomas NR,O'Shaughnessy MM,O'Regan JA,et al.C3 glomerulopathy:clinicopathologic features and predictors of outcome.Clin JAm Soc Nephrol,2014,9(1):46-53.
[107]Medjeral-Thomas N,Malik TH,PatelMP,et al.A novel CFHR5 fusion protein causes C3 glomerulopathy in a family without Cypriot ancestry.Kidney Int,2014,85(4):933-937.
[108]Lu DF,Moon M,Lanning LD,et al.Clinical features and outcomes of 98 children and adultswith dense deposit disease.Pediatr Nephrol,2012,27(5):773-781.
[132]Padwal RJ,Hemmelgam BR,Khan NA,et al.The 2008 Canadian Hypetension Education Program recommendations for the management of hypertension:part 1-blood pressure meuremem,diagnosis and assessment of risk.Can JCardiol,2008,24(6):455-463.
[133]Mazzali M,Hugles J,Kim YG,et al.Elevated uric acid increases blood pressure in the rat by a novel crystal-independentmechanism.Hypertension,2001,38(5):1101-1106.
[134]Peraha CA,Shlipak MG,Judd S,et al.Detection of chronic kidney disease with ereatinine,eystatin C,and urine albumin to cereatinine ratio and association with progression to end-stage renal disease and mortality.JAMA,2011,305(15):1545-1552.
[135]Yashiro M,Kamata T,Segawa H,et al.Comparisons of cystatin C with creatinine for evaluation of renal function in chronic kidney disease.Clin Exp Nephrol,2009,13(6):598-604.
[136]Feig DI,Kang DH,Nakagawa T,et al.Uric acid and hypertension.Curt Hypertens Rep,2006,8(2):111-115.
[137]Kal HB,van Kempen-Harteveld ML.Renal dysfunction after total body irradiation:dose-effect relationship.Int JRadiat Oncol Biol Phys,2006,65(4):1228.
[138]Cohen EP,Moulder JE,Robbins ME.Radiation nephropathy caused by yttrium 90.Lancet,2001,358(9287):1102-1103.
[139]Zenz T,Schlenk RF,Glatting G,et al.Bone marrow transplantation nephropathy after an intensified conditioning regimen with radioimmunotherapy and allogeneic stem cell transplantation.J Nucl Med,2006,47(2):278-286.
[140]Lambert B,Cybulla M,Weiner SM,et al.Renal toxicity after radionuclide therapy.Radiat Res,2004,161(5):607-611.
[141]Juncos LI,Carrasco Due?as S,Cornejo JC,etal.Long-term enalapril and hydrochlorothiazide in radiation nephritis.Nephron,1993,64(2):249-255.
[142]Grassegger A,Pohla-Gubo G,Frauscher M,et al.Autoantibodies in systemic sclerosis(scleroderma):clues for clinical evaluation,prognosis and pathogenesis.Wien Med Wochenschr,2008,158(1-2):19-28.
[143]Gussin HA,Ignat GP,Varga J,et al.Anti-topoisomeraseⅠ(anti-Scl-70)antibodies in patients with systemic lupus erythematosus.Arthritis Rheum,2001,44(2):376-383.
[144]Cepeda E J,Reveille JD.Autoantibodies in systemic sclerosis and fibrosing syndromes:clinical indications and relevance.Curt Opin Rheumatol,2004,16(6):723-732.
[145]Henault J,Robitaille G,Senecal JL,et al.DNA topoisomeraseⅠbinding to fibroblasts induces monocyte adhesion and activation in the presence of anti-topoisomeraseⅠautoantibodies from systemic sclerosis patients.Arthritis Rheum,2006,54(3):963-973.
[146]Henault J,Tremblay M,Clement I,et al.Direct binding of anti-DNA topoisomeraseⅠautoantibodies to the cell surface of fibro-blasts in patients with systemic sclerosis.Arthritis Rheum,2004,50(10):3265-3274.
[154]Rej S,Elie D,Mucsi I,et al.Chronic kidney disease in lithiumtreated older adults:a review of epidemiology,mechanisms,and implications for the treatment of late-life mood disorders.Drugs aging,2015,32(1):31-42.
[155]Johnson G.Lithium early development,toxicity,and renal function.Neuropsychopharmacology,1998,19(3):200-205.
[156]Meyem CM.Acute and Chronic Tubulointerstitial Disease.In:Arthur Greenberg.Primer on Kidney DiseasesM.4th ed.National Kidney Foundation,2005.
[157]Temiz Y,Tarcan T,Onol FF,et al.The efficacy of Tc99m dimercaptosuccinic acid(Tc-DMSA)scintigraphy and ultrasonography in detecting renal scars in children with primary vesicoureteral reflux(VUR).Int Urol Nephrol,2006,38(1):149-152.
[158]Kamisawa T,Funata N,Hayashi Y,et al.A new clinicopathological entity of IgG4-related autoimmune disease.JGastroenterol,2003,38(10):982-984.
[159]Shimosegawa T,Chari ST,Frulloni L,et al.International consensus diagnostic criteria for autoimmune pancreatitis:guidelines of the International Association of Pancreatology.Pancreas,2011,40(3):352-358.
[160]Kawa S,Hamano H.Autoimmune pancreatitis and bile duct lesions.J Gastroenterol,2003,38(12):1201-1203.
[161]Culver EL,Bateman AC.IgG4-related disease:can non-classical histopathological features or the examination of clinically uninvolved tissues be helpful in the diagnosis?JClin Pathol,2012,65(11):983-968.
[162]Cortazar FB,Stone JH.IgG4-related disease and the kidney.Nat Rev Nephrol,2015,11(10):599-609.
[163]Deshpande V,Zen Y,Chan JK,et al.Consensus statement on the pathology of IgG4-related disease.Mod Pathol,2012,25(9):1181-1192.
[164]Pradhan D,Pattnaik N,Silowash R,et al.IgG4-related kidney disease-A review.Pathol Res Pract,2015,211(10):707-711.
[165]Vikram D,Wallace ZS,Crowe JL,et al.International Consensus Guidance Statement on the Managementand Treatmentof IgG4-Related Disease.Arthritis Rheumatol,2015,67(7):1688-1699.